Browsing by Author "Labiano Ciriza, Ibone"

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      The role of TREM-2 receptor in cholestasis and fibrosis-associated hepatocellular carcinoma (HCC) 

      Labiano Ciriza, Ibone (2020-12-18)
      La progresión de enfermedades hepatobiliares crónicas, tales como las enfermedades colestásicas (entrelas que destacan la colangitis biliar primaria y la colangitis esclerosante primaria), así como el carcinomahepatocelular ...
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      TREM-2 defends the liver against hepatocellular carcinoma through multifactorial protective mechanisms 

      Esparza Baquer, Aitor ORCID; Labiano Ciriza, Ibone; Sharif, Omar; Agirre Lizaso, Aloña; Oakley, Fiona; Rodrigues, Pedro M.; Zhuravleva, Ekaterina; O'Rourke, Colm J.; Hijona Muruamendiaraz, Elizabeth; Jiménez Agüero, Raúl; Riaño Fernández, Ioana; Landa Magdalena, Ana; La Casta, Adelaida; Zaki, Marco Y. W.; Muñoz, Patricia; Azkargorta, Mikel; Elortza, Felix; Vogel, Andrea; Schabbauer, Gernot; Aspichueta Celaá, Patricia; Andersen, Jesper B.; Knapp, Sylvia; Mann, Derek A.; Bujanda Fernández de Pierola, Luis ORCID; Bañales Asurmendi, Jesús María ORCID; Perugorria Montiel, María Jesús (BMJ Publishing Group, 2021-06-07)
      [EN] Objective Hepatocellular carcinoma (HCC) is a prevalent and aggressive cancer usually arising on a background of chronic liver injury involving inflammatory and hepatic regenerative processes. The triggering receptor ...
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      TREM-2 plays a protective role in cholestasis by acting as a negative regulator of inflammation 

      Labiano Ciriza, Ibone; Agirre Lizaso, Aloña; Olaizola Rebe, Paula; Echebarria, Anne; Huici Izagirre, Maider; Olaizola, Irene; Esparza Baquer, Aitor ORCID; Sharif, Omar; Hijona Muruamendiaraz, Elizabeth; Milkiewicz, Piotr; Milkiewicz, Malgorzata; González Romero, Francisco; Aspichueta Celaá, Patricia; Monte, María J.; García Marín, Jose Juan; Vucur, Michael; Luedde, Tom; Marzioni, Marco; Mann, Derek A.; Bujanda Fernández de Pierola, Luis ORCID; Perugorria Montiel, María Jesús (Elsevier, 2022-10)
      Background & Aims: Inflammation, particularly that mediated by bacterial components translocating from the gut to the liver and binding to toll-like receptors (TLRs), is central to cholestatic liver injury. The triggering ...