BackBrowsing by Author "Rabe, Sabine"
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Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology.
; ; ; ; ; ; Capetillo González de Zarate, Estíbaliz
; ; ; ; ; (Society for Neuroscience, 2012-01)
An early role of amyloid-beta peptide (Abeta) aggregation in Alzheimer’s disease pathogenesis is well established. However, the contribution of intracellular or extracellular forms of Abeta to the neurodegenerative process ...